Fig. 5From: Up-regulation of DcR3 in microbial toxins-stimulated HUVECs involves NF-κB signallingNF-κB signalling pathway is involved in endogenous DcR3 expression in HUVECs stimulated by LPS, LTA and zymosan. TLR-2 and TLR-4 utilize MyD88-adaptor-like (MAL), which lead to the activation of MAPKs and NF-κB. The active NF-κB promotes DcR3 secretionBack to article page